Alkaline diet (also known as the alkaline ash diet, alkaline acid diet, acid ash diet, and the acid alkaline diet) describes a group of loosely related diets based on the belief that certain foods can affect the acidity and pH of bodily fluids, including the urine or blood, and can therefore be used to treat or prevent diseases. Due to the lack of human studies supporting any benefits of this diet, it is generally not recommended by dieticians and other health professionals.[1]

The relationship between diet and acid-base homeostasis, or the regulation of the acid-base status of the body, has been studied for decades, though the medical applications of this hypothesis have largely focused on changing the acidity of urine. Traditionally, this diet has advocated for avoiding meat, poultry, cheese, and grains in order to make the urine more alkaline (higher pH), changing the environment of the urine to prevent recurrent urinary tract infections (UTIs) and kidney stones (nephrolithiasis). However, difficulties in effectively predicting the effects of this diet have led to medications, rather than diet modification, as the preferred method of changing urine pH. The "acid-ash" hypothesis has been considered a risk factor for osteoporosis by various scientific publications, though more recently, the available weight of scientific evidence does not support this hypothesis.

The term "alkaline diet" has also been used by alternative medicine practitioners, with the proposal that such diets treat or prevent cancer, heart disease, low energy levels as well as other illnesses. These claims are not supported by medical evidence and make incorrect assumptions about how alkaline diets function that are contrary to modern understanding of human physiology.

According to the traditional hypothesis underlying this diet, acid ash is produced by meat, poultry, cheese, fish, eggs, and grains. Alkaline ash is produced by fruits and vegetables, except cranberries, prunes and plums. Since the acid or alkaline ash designation is based on the residue left on combustion rather than the acidity of the food, foods such as citrus fruits that are generally considered acidic are actually considered alkaline producing in this diet.[2]

It has been suggested that diets high in "acid ash" (acid producing) elements will cause the body to try to buffer (or counteract) any additional acid load in the body by breaking down bone, leading to weaker bones and increased risk for osteoporosis. Conversely, "alkaline ash" (alkaline producing) elements will hypothetically decrease the risk of osteoporosis. This hypothesis has been advanced in a position statement of the American Dietetic Association,[2] in a publication of the U.S. National Academy of Sciences,[3] as well as other scientific publications,[4] which have stated foods high in potassium and magnesium such as fruits and vegetables may decrease the risk of osteoporosis through increased alkaline ash production. This acceptance of the acid-ash hypothesis as a major modifiable risk factor of osteoporosis by these publications, however, was largely made without significant critical review by high quality systematic analysis.[5]

Recent systematic reviews have been published which have methodically analyzed the weight of available scientific evidence, and have found no significant evidence to support the acid-ash hypothesis in regards to prevention of osteoporosis. A meta-analysis of studies on the effect of dietary phosphate intake contradicted the expected results under the acid-ash hypothesis with respect to calcium in the urine and bone metabolism. This result suggests use of this diet to prevent calcium loss from bone is not justified.[5] Other meta-analyses which have investigated the effect of total dietary acid intake have also found no evidence that acid intake increases the risk for osteoporosis as would be expected under the acid-ash hypothesis.[4][6] A review looked at the effects of dairy product intake, which have been hypothesized to increase the acid load of the body through phosphate and protein components. This review found no significant evidence suggesting dairy product intake causes acidosis or increases risk for osteoporosis.[7]

It has also been speculated that this diet may have an effect on muscle wasting, growth hormone metabolism or back pain, though there is no conclusive evidence to confirm these hypotheses.[8][9][10] Given an aging population, the effects of an alkaline diet on public health may be worth considering, though there is little scientific evidence in this area.[10]

Alternative medicine practitioners who have promoted the alkaline diet have advocated its use in the treatment of various medical conditions including cancer.[11] These claims have been mainly promoted on websites, magazines, direct mail, and books, and have been mainly directed at a lay audience.[4] While it has been proposed that this diet can help increase energy, lose weight, and treat cancer and heart disease, there is no evidence to support any of these claims.[12] This version of the diet, in addition to avoiding meats and other proteins, also advocates avoiding processed foods, white sugar, white flour, and caffeine,[9] and can involve specific exercise and nutritional supplement regimens as well.[13]

Advocates for alternative uses of an alkaline diet propose that since the normal pH of the blood is slightly alkaline, the goal of diet should be to mirror this by eating a diet that is alkaline producing as well. These advocates propose that diets high in acid-producing elements will generally lead the body to become acidic, which can foster disease.[9][12] This proposed mechanism, in which the diet can significantly change the acidity of the blood, goes against "everything we know about the chemistry of the human body" and has been called a "myth" in a statement by the American Institute for Cancer Research.[14] Unlike the pH level in the urine, a selectively alkaline diet has not been shown to elicit a sustained change in blood pH levels, nor to provide the clinical benefits claimed by its proponents. Because of the body's natural regulatory mechanisms, which do not require a special diet to work, eating an alkaline diet just can, at most, change the blood pH minimally and transiently.[1][9][12][14]

A similar proposal by those advocating this diet suggests that cancer grows in an acidic environment, and that a proper alkaline diet can change the environment of the body to treat cancer. This proposal ignores the fact that while cancer tissue does grow in acidic environment, it is the cancer that creates the acidity. The rapid growth of cancer cells creates the acidic environment; the acidic environment does not create cancer.[11] The proposal also neglects to recognize that it is "virtually impossible" to create a less acidic environment in the body.[14] "Extreme" dietary plans such as this diet have more risks than benefits for patients with cancer.[11]

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Alkaline diet - Wikipedia, the free encyclopedia

Welcome! If you want to lose weight, gain muscle, increase energy levels or just generally look and feel healthier you've come to the right place.

Here's where to start:

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A couple weeks back, I wrote about the top 8 most common reactions you get when people hear you dont eat grains, and I offered up some concise responses to those reactions. It was well received, so I thought Id do the same thing for your high-fat diet. If you thought having to explain your grain-free diet was tough, explaining a high-fat diet in particular, a high-animal fat diet may seem even harder. At least with a grain-free diet, youre merely removing something that many hold near and dear to their hearts. Its healthy and delicious, sure, but at least youre not adding something that will actively kill you. Fat is that deadly thing, for many people. Its fat, for crying out loud. Its bad for you, practically a poison. Everyone knows it. I mean, have you seen what fat down the kitchen drain does to your plumbing?

Actually, like the grain-free diet, explaining the high-fat diet is not that hard. Ill even promise you that there are ways to do it, explanations and answers that dont make you seem like a crazy person who hates his heart (I make no such promises for those of you with a stick of butter with bite marks and a tub of coconut oil with a greasy spoon beside it on your office desk, however). Now lets get right to their questions and responses you can use:

Isnt all that fat gonna glom onto your arteries?

That isnt how it works. Atherosclerosis is caused by oxidized LDL particles penetrating the arterial wall, inciting inflammation, and damaging the arterial tissue. It is not caused by fat mechanistically attaching itself to the surface of the arteries like fat in a kitchen pipe. Also, its not like you eat some butter and that butter gets directed straight into your bloodstream. Your blood doesnt have oil slicks running through it, or congealed droplets of grease gumming up the passageways. You are the product of millions upon millions of years of evolution, and I think our bodies can do better than trying to ape modern plumbing.

Response: My arteries are not pipes. Fat is not solidifying in my blood like it can in the plumbing. Atherosclerosis is a complex process with dozens of factors beyond whats in your diet, let alone the fat content.

Isnt all that cholesterol gonna raise your cholesterol?

If I were a rabbit, sure. When you feed cholesterol to an herbivorous animal, like a rabbit, whose only encounters with dietary cholesterol occur in a lab setting, their blood lipids will increase and they will usually develop atherosclerosis. For many years, the cholesterol-fed rabbit was a popular model for studying heart disease and gave rise to the now-popular idea that dietary cholesterol also elevates blood lipids in humans (thus immediately condemning them to a heart attack, naturally). Except it isnt the case. Save for a select few who are hyper-responders, the vast majority of people can eat cholesterol without it affecting their cholesterol levels. And even when dietary cholesterol affects blood lipids, its usually an improvement, increasing HDL and the HDL:TC ratio while leaving LDL mostly unchanged.As for where all that blood cholesterol comes from, we make pretty much all the cholesterol in our blood in-house, and dietary cholesterol tends to suppress endogenous cholesterol synthesis. Boy, between staying local and only making as much as we need, our livers are downright green. I bet our HDL is GMO-free and organic to boot (not so sure about those sneaky LDL particles, though).

Continue reading here:
Why A High-Fat Diet is Healthy and Safe | Mark's Daily Apple

Diabetic diet refers to the diet that is recommended for people with diabetes mellitus. There is much controversy regarding what that diet should consist of. The diet most often recommended is high in dietary fiber, especially soluble fiber, but low in fat (especially saturated fat) and low in sugar. Recommendations of the fraction of total calories to be obtained from carbohydrate are generally in the range of 40 to 65%, but recommendations can vary as widely as from 16 to 75% .[1] People with diabetes may be encouraged to reduce their intake of carbohydrates that have a high glycemic index (GI), although this is also controversial.[2] (In cases of hypoglycemia, they are advised to have food or drink that can raise blood glucose quickly, such as lucozade, followed by a long-acting carbohydrate (such as rye bread) to prevent risk of further hypoglycemia.) However, others question the usefulness of the glycemic index and recommend high-GI foods like potatoes and rice. It has been claimed that oleic acid has a slight advantage over linoleic acid in reducing plasma glucose.[3]

There has been long history of dietary treatment of diabetes mellitus dietary treatment of diabetes mellitus was used in Egypt since 3,500 B.C., and was used in India by Sushruta and Charaka more than 2000 years ago. In the eighteenth century, John Rollo argued that calorie restriction in the diabetic diet could reduce glycosuria in diabetes. However, more modern history of the diabetic diet may begin with Frederick Madison Allen, who, in the days before insulin was discovered, recommended that people with diabetes ate only a low-calorie diet to prevent ketoacidosis from killing them. This was an approach which did not actually cure diabetes but merely extended life by a limited period. The first use of insulin by Frederick Banting in 1922 changed things, and at last allowed patients more flexibility in their eating.

In the 1950s, the American Diabetes Association, in conjunction with the U.S. Public Health Service, introduced the "exchange scheme". This allowed people to swap foods of similar nutritional value (e.g. carbohydrate) for another, so, for example, if wishing to have more than normal carbohydrates for dessert, one could cut back on potatoes in one's first course. The exchange scheme was revised in 1976, 1986 and 1995.[4]

However, not all diabetes dietitians today recommend the exchange scheme. Instead, they are likely to recommend a typical healthy diet: one high in fiber, with a variety of fruit and vegetables, and low in both sugar and fat, especially saturated fat. A diet that is high in plant fibre was recommended by James Anderson (Anderson & Ward, 1979; cited in Murray & Pizzorno, 1990). This may be understood as continuation of the work of Denis Burkitt and Hugh Trowell on dietary fibre,[5] which in turn, may be understood as a continuation of the work of Price (Murray & Pizzorno, 1990). It is still recommended that people with diabetes consume a diet that is high in dietary fiber.

In 1976, Nathan Pritikin opened a centre where patients were put on programme of diet and exercise (the Pritikin Program). This diet is high on carbohydrates and fibre, with fresh fruit, vegetables, and whole grains. A study at UCLA in 2005 showed that it brought dramatic improvement to a group of people with diabetes or pre-diabetes in only three weeks, so that about half no longer met the criteria for the disease.[6][7][8][9]

On the other hand, in 1983, Richard K. Bernstein began treating people with diabetes and pre-diabetes successfully with a very low carbohydrate diet, avoiding fruit, added sugar, and starch. Both the Pritikin approach and the Bernstein approach prescribe exercise.

An approach that has been popular with some people with type one diabetes mellitus since 2000 is known as DAFNE (Dose Adjustment for Normal Eating). This approach involves estimating how much carbohydrates there will be in a meal and modifying the amount of insulin one injects before meal accordingly. An equivalent approach has for people with type two diabetes mellitus is known as DESMOND, which stands for Diabetes Education and Self-Management for On-Going and Newly Diagnosed (diabetes). DAFNE has its own newsletter and has received recommendation.[10]

The American Diabetes Association in 1994 recommended that 6070% of caloric intake should be in the form of carbohydrates. As mentioned above, this is controversial, with some researchers claiming that 40%[11] or even less is better, while others claim benefits for a high-fiber, 75% carbohydrate diet.[12]

An article summarizing the view of the American Diabetes Association[13] contains the statement: "Sucrose-containing foods can be substituted for other carbohydrates in the meal plan or, if added to the meal plan, covered with insulin or other glucose-lowering medications. Care should be taken to avoid excess energy intake." Sucrose does not increase glycemia more than the same number of calories taken as starch[citation needed]. It is not recommended to use fructose as a sweetener. Benefits may be obtained by consumption of dietary fibre in conjunction with carbohydrate; as Francis (1987) points out, evidence suggests that carbohydrate consumed with dietary fiber will have a less major impact on glycemic rise than the same amount of carbohydrate consumed alone.

What has not generally been included in diabetic diet recommendations is the variation in effect from different carbohydrates. It has been recommended that carbohydrates eaten by people with diabetes should be complex carbohydrates.

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Diabetic diet - Wikipedia, the free encyclopedia

If you need to learn how Irritable Bowel Syndrome symptoms (pain, diarrhea, constipation, bloating) can be controlled through an IBS diet plan and eating IBS recipes, you're in the right place.

Despite the fact that diet plays a direct role in gut function (which is instinctively obvious to IBS sufferers, who are desperate for a reliable IBS diet as they know this will help them), many doctors fail to give their patients any IBS dietary or food guidelines for Irritable Bowel Syndrome treatment at all.

Worse still, much IBS dietary information available for Irritable Bowel Syndrome is outdated and useless - or likely to trigger IBS symptoms (have you been told to eat wheat bran? lots of raw veggies for fiber? this is wrong!).

The proper Irritable Bowel Syndrome diet plan (and yes, there is one - IBS is not so highly individualized that no accurate generalizations can be made) makes a world of difference for almost everyone with Irritable Bowel Syndrome.

Share the IBS Diet!

You likely already know from personal experience that some foods nearly always cause IBS symptoms, while others never seem to bother you. On the other hand, you've also probably noticed that sometimes a specific food will trigger an Irritable Bowel Syndrome attack, while at other times you can eat the exact same thing without difficulty. Odds are it doesn't seem like there's any rhyme or reason to this. Odds are also that you've been wracking your brain to figure out why.

There are, in fact, very clear IBS dietary guidelines to follow for how to eat safely for Irritable Bowel Syndrome, based on the well-established effects certain categories of foods have on the GI tract. The key word here is categories most people with IBS drive themselves bonkers trying to find that one specific food that is triggering their Irritable Bowel Syndrome.

The problem is, it isn't a single food that causes Irritable Bowel Syndrome symptoms. It's ANY food that is high in fat, insoluble fiber, caffeine, coffee (even decaf), carbonation, or alcohol. Why? Because all of these food categories are either GI stimulants or irritants, and can cause violent reactions of your gastrocolic reflex. This directly affects the muscles in your colon and can lead to IBS pain, constipation AND diarrhea, gas, and bloating.

In fact, the happy truth is that eating safely for Irritable Bowel Syndrome does not mean deprivation, never going to restaurants, bland food, or an unhealthily limited diet. Nor does it mean living on "rabbit food" available exclusively at health food stores, or following brutal elimination diets, or keeping endless IBS food diaries for the rest of your life.

It does mean learning to eat safely by realizing how different foods physically affect the GI tract, and how the same foods can help or hurt both Irritable Bowel Syndrome diarrhea AND constipation, as well as bloating, gas, nausea, and painful abdominal cramps. Following the Irritable Bowel Syndrome diet simply means learning how foods can prevent or trigger a spastic colon.

Read more here:
IBS Diet, Irritable Bowel Syndrome Diet ~ The Eating for ...

10/8/2007

JUDDDD, or the Johnson Up Day Down Day Diet. I've been doing it for over 3 months. I lost 9 lbs in the first 3 weeks. I went on to lose another 6 lbs more gradually and have since gained 3 lbs back (I'm on maintenance now, essentially). I do not exercise (injuries).

I was thin to begin with (that 9 lbs was nearly 9% of my starting weight), too.

It's a great diet because it is so very easy. You eat every other day. It can be done different ways, however. For optimal results, eat nothing (no calories) on down days, and stay within 200 cals of your recommended caloric intake (to maintain your current weight) on your up days. I did complete fasting on my down days for the first 6 weeks.

Many people can't fast completely, so for them, you'd do 20-30% (again, of recommended caloric intake to maintain). That ranges from 300-800 calories depending on your starting weight and activity level. I recommend doing shakes or bars on your down days if you suck at calorie counting or are likely to binge.

Now I do about 20% on my down days because I'm happy with the weight I've at. But I tend to eat close to double my recommended calories on up days, because OMG NY FOOD. I'd probably stay at my lower weight or keep losing if I actually ate normally on my up days.

The official website (Dr. Johnson is awesome): johnsonupdaydowndaydiet.

I wrote a whole FAQ on it: community.livejournal.co

Good luck, feel free to ask questions!

See the original post:
Quickest way to lose 10 pounds? Share your gimmick diets ...

This article is about a dietary therapy for epilepsy. For information on ketogenic low-carbohydrate diets as a lifestyle choice or for weight loss, see Low-carbohydrate diet.

The ketogenic diet is a high-fat, adequate-protein, low-carbohydrate diet that in medicine is used primarily to treat difficult-to-control (refractory) epilepsy in children. The diet forces the body to burn fats rather than carbohydrates. Normally, the carbohydrates contained in food are converted into glucose, which is then transported around the body and is particularly important in fuelling brain-function. However, if there is very little carbohydrate in the diet, the liver converts fat into fatty acids and ketone bodies. The ketone bodies pass into the brain and replace glucose as an energy source. An elevated level of ketone bodies in the blood, a state known as ketosis, leads to a reduction in the frequency of epileptic seizures.[1]

The original therapeutic diet for paediatric epilepsy provides just enough protein for body growth and repair, and sufficient calories[Note 1] to maintain the correct weight for age and height. This classic ketogenic diet contains a 4:1 ratio by weight of fat to combined protein and carbohydrate. This is achieved by excluding high-carbohydrate foods such as starchy fruits and vegetables, bread, pasta, grains and sugar, while increasing the consumption of foods high in fat such as nuts, cream and butter.[1]

Most dietary fat is made of molecules called long-chain triglycerides (LCTs). However, medium-chain triglycerides (MCTs)made from fatty acids with shorter carbon chains than LCTsare more ketogenic. A variant of the classic diet known as the MCT ketogenic diet uses a form of coconut oil, which is rich in MCTs, to provide around half the calories. As less overall fat is needed in this variant of the diet, a greater proportion of carbohydrate and protein can be consumed, allowing a greater variety of food choices.[2][3]

The classic therapeutic ketogenic diet was developed for treatment of paediatric epilepsy in the 1920s and was widely used into the next decade, but its popularity waned with the introduction of effective anticonvulsant drugs. In the mid-1990s, Hollywood producer Jim Abrahams, whose son's severe epilepsy was effectively controlled by the diet, created the Charlie Foundation to promote it. Publicity included an appearance on NBC's Dateline programme and ...First Do No Harm (1997), a made-for-television film starring Meryl Streep. The foundation sponsored a multicentre research study, the results of whichannounced in 1996marked the beginning of renewed scientific interest in the diet.[1]

Almost half of children and young people with epilepsy who have tried some form of this diet saw the number of seizures drop by at least half, and the effect persists even after discontinuing the diet.[4] The most common adverse effect is constipation, affecting about 30% of patientsthis was due to fluid restriction, which was once a feature of the diet, but this led to increased risk of kidney stones, and is no longer considered beneficial.[4][5] There is some evidence that adults with epilepsy may benefit from the diet, and that a less strict regimen, such as a modified Atkins diet, is similarly effective.[1] Clinical trials and studies in animal models (including C. elegans[6]) suggest that ketogenic diets provide neuroprotective and disease-modifying benefits for a number of adult neurodegenerative disorders.[7][8] As of 2012, there is limited clinical trial data in these areas, and, outside of paediatric epilepsy, use of the ketogenic diet remains at the research stage.[5][9][10]

Epilepsy is one of the most common neurological disorders after stroke,[11] and affects at least 50million people worldwide.[12] It is diagnosed in a person having recurrent unprovoked seizures. These occur when cortical neurons fire excessively, hypersynchronously, or both, leading to temporary disruption of normal brain function. This might affect, for example, the muscles, the senses, consciousness, or a combination. A seizure can be focal (confined to one part of the brain) or generalised (spread widely throughout the brain and leading to a loss of consciousness). Epilepsy may occur for a variety of reasons; some forms have been classified into epileptic syndromes, most of which begin in childhood. Epilepsy is considered refractory (not yielding to treatment) when two or three anticonvulsant drugs have failed to control it. About 60% of patients will achieve control of their epilepsy with the first drug they use, whereas about 30% do not achieve control with drugs. When drugs fail, other options include epilepsy surgery, vagus nerve stimulation and the ketogenic diet.[11]

The ketogenic diet is a mainstream therapy that does not use pharmaceutical drugs, which was developed to reproduce the success and remove the limitations of the non-mainstream use of fasting to treat epilepsy.[Note 2] Although popular in the 1920s and 30s, it was largely abandoned in favour of new anticonvulsant drugs.[1] Most individuals with epilepsy can successfully control their seizures with medication. However, 2030% fail to achieve such control despite trying a number of different drugs.[9] For this group, and for children in particular, the diet has once again found a role in epilepsy management.[1][13]

Physicians of ancient Greece treated diseases, including epilepsy, by altering their patients' diet. An early treatise in the Hippocratic Corpus, On the Sacred Disease, covers the disease; it dates from c.400 BC. Its author argued against the prevailing view that epilepsy was supernatural in origin and cure, and proposed that dietary therapy had a rational and physical basis.[Note 3] In the same collection, the author of Epidemics describes the case of a man whose epilepsy is cured as quickly as it had appeared, through complete abstinence of food and drink.[Note 4] The royal physician Erasistratus declared, "One inclining to epilepsy should be made to fast without mercy and be put on short rations."[Note 5]Galen believed an "attenuating diet"[Note 6] might afford a cure in mild cases and be helpful in others.[14]

The first modern study of fasting as a treatment for epilepsy was in France in 1911.[15] Twenty epilepsy patients of all ages were "detoxified" by consuming a low-calorie vegetarian diet, combined with periods of fasting and purging. Two benefited enormously, but most failed to maintain compliance with the imposed restrictions. The diet improved the patients' mental capabilities, in contrast to their medication, potassium bromide, which dulled the mind.[16]

Continued here:
Ketogenic diet - Wikipedia, the free encyclopedia

I have a gut feeling that things are about to become much more interesting in the science world. As researchers continue to discover more and more exciting news about just how our microbiomes can uniquely identify us, change our food cravings, and

Wouldnt you enjoy taking it easy once in awhile? Our fast-paced, adrenalized modern lifestyle is mismatched with our Stone Age genes.1 Eating and moving like a hunter-gatherer are fundamental changes to support our physiology. However, there are additional ways to optimize our gene expression2 and mitigate the ill effects on constantly being on the go []

The marathon and triathlon seasons are fully underway and this year over half a million people will complete the marathon and thousands more participate in triathlons and other endurance events. Whether theyre entering an event for the first time or trying to achieve a personal best time, one of the most common questions I get []

Introduction: Evolutionary Perspective Its pretty clear that if we follow the example of our hunter gatherer ancestors, artificial sweeteners should not be part of contemporary Stone Age diets. In my book, The Paleo Diet Revised (2010)1 I warned against drinking artificially sweetened soft drinks and further strengthened my opposition to all artificial sweeteners in 2012 []

Over the past 5-7 years, more and more people worldwide have become aware of the Paleo Diet, which really is not a diet at all, but rather a lifelong way of eating to reduce the risk of chronic disease and maximize health and wellbeing. One of the fundamental principles of The Paleo Diet is to []

Over the years since the publication of my first book, I have been asked time and again if there is a vegetarian version of The Paleo Diet. Ive got to say emphatically No! Vegetarian diets are a bit of a moving target because they come in at least three major versions. We all know []

I have noticed in the last few years that many Paleo Dieters believe that potatoes can be regularly consumed without any adverse health effects. Part of this misinformation seems to stem from writers of blogs and others who are unfamiliar with the scientific literature regarding potatoes. So should we be eating potatoes or not?

The Recent Evolutionary Introduction of Milk and Dairy One of the rewarding benefits of having written a diet book that has become internationally known is the opportunity to travel the world and speak to tens of thousands of people about this engaging and life changing subject. My signature lecture, Origins and Evolution of the Western []

With the plethora of benefits supported by scientific evidence,1 Gluten-free diets have been gaining in popularity in recent years.2 Studies range from gastrointestinal symptom improvement,3 to possible correlations with autism,4 and diabetes.5 However, there may not be a more fascinating area of gluten study than how the protein composite can be related to cognitive function.6 []

Over the past few years, craze over bacon has surged in the Paleo community, but is it Paleo or isnt it? It seems like just about every bacon issue under the sun has been argued, discussed and disputed on Paleo blogs, websites and cookbooks. I have little to add to these specific discussions because as []

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The Paleo Diet - Live Well, Live Longer.

Volunteers have created a package of materials designed to make the diet as easy and effective as possible.

Our Foodlist and Handbook will guide you step-by-step as you test out the Program, and we will keep you up-to-date about product changes via "Product Alert" emails and in our newsletter, Pure Facts.

There are many volunteers who are here to help you with any questions. You can reach us via email, phone, or through our popular members' Facebook.

Get a sneak peek at what members get by clicking on More

Happily, there is a huge selection of foods of every kind, including snacks, desserts, convenience foods and mixes that are acceptable on the Feingold Diet. More

Who was Dr. Feingold? Chief of Allergy and Chairman of the Central Research Committee, Kaiser Foundation Hospitals of Northern California. More

Who we are and why we created the Feingold Association.

We really tried to do what was best for our child. We looked forward to welcoming our new baby into the world, having read books and articles and talked with friends about how to give a our new little one the best start in life. But it didn't work out the way we had planned... More

Meet "Our Kids"

Geoffrey was a 7-year-old who would fly into a rage over nothing ...

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The Feingold Diet Program for ADHD